Here you will find some interesting articles and lecture transcripts to read.
NEURO VASCULAR BUNDLE RELATED:
Initially planned for this conference meeting was a one-dayworkshop
with Christoph Sommer and Konrad Obermeier on Thursday, April 15th 2021
“Bridging the developmental movement of the embryo with structural pathologies”.
Due to covid-related changes this plan morphed into a zoom event
on Saturday, April 17th 2021. Below you find the Text-Version of this
zoom-event-contribution by: Konrad Obermeier
“Leg development and the neuro-vascular-bundle (NVB)”.
A neuro-vascular-bundle (NVB) embryologically functions as a
developmental fulcrum guiding the growth-movement of
Structure and is morphologically contributing to manifesting
form. A NVB is wrapped in layered sheets of connective
tissues. We can manually address these original and central
tensional pathways, for example in the relationship of the
legs to the lumbar spine.
Epi-Perineurial Anatomy, Innervation, and Axonal Nociceptive Mechanisms
Geoffrey Bove, DC, PhD
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 01907
Published in final edited form as:
J Bodyw Mov Ther. 2008 July ; 12(3): 185–190. doi:10.1016/j.jbmt.2008.03.004.
Body workers from all disciplines appreciate that maneuvers that move nerves often reproduce
radiating pain. This symptom reproduction has important implications for the diagnosis and
management of radiating pain symptoms.
In the presentation at Fascia 2007 from which this manuscript is derived, two videos that were
obtained with high resolution diagnostic ultrasound were presented that clearly showed median
nerve gliding during normal finger and wrist movements. The movements were independent
of the movements of the other surrounding structures. Such movements of the interface between
the nerve and the surrounding structures constitute but one mechanical stimulus that nerves
are susceptible to. Nerves are also bent around various structures, and indented by external
pressures. Nerves have many anatomical features that allow them to accommodate such
movements and mechanical stimuli. The reader is directed to books by Shacklock (Shacklock
2005) and Butler (Butler 2000) for full descriptions of nerve biomechanics.1
Intramuscular Connective Tissue Differences in Spastic
and Control Muscle: A Mechanical and Histological Study
Marije de Bruin1, Mark J. Smeulders1, Michiel Kreulen1,2, Peter A. Huijing3, Richard T Jaspers3*
1 Department of Plastic, Reconstructive and Hand Surgery, Academic Medical Center, Amsterdam, The Netherlands,
2 Department of Plastic, Reconstructive and Hand
Surgery, Red Cross Hospital, Beverwijk, The Netherlands,
3 Laboratory for Myology, MOVE Research Institute Amsterdam, Faculty of Human Movement Sciences, VU University Amsterdam, Amsterdam, The Netherlands
Cerebral palsy (CP) of the spastic type is a neurological disorder characterized by a velocity dependent increase in tonic stretch reflexes with exaggerated tendon jerks. Secondary to the spasticity, muscle adaptation is presumed to contribute to limitations in the passive range of joint motion. However, the mechanisms underlying these limitations are unknown. Using
biopsies, we compared mechanical as well as histological properties of flexor carpi ulnaris muscle (FCU) from CP patients (n = 29) and healthy controls (n = 10). The sarcomere slack length (mean 2.5 mm, SEM 0.05) and slope of the normalized sarcomere length-tension characteristics of spastic fascicle segments and single myofibre segments were not different from those of control muscle. Fibre type distribution also showed no significant differences. Fibre size was significantly smaller
(1933 mm2, SEM 190) in spastic muscle than in controls (2572 mm2, SEM 322). However, our statistical analyses indicate that the latter difference is likely to be explained by age, rather than by the affliction. Quantities of endomysial and perimysialnetworks within biopsies of control and spastic muscle were unchanged with one exception: a significant thickening of the
tertiary perimysium (3-fold), i.e. the connective tissue reinforcement of neurovascular tissues penetrating the muscle. Note that this thickening in tertiary perimysium was shown in the majority of CP patients, however a small number of patients (n = 4 out of 23) did not have this feature. These results are taken as indications that enhanced myofascial loads on FCU is one among several factors contributing in a major way to the aetiology of limitation of movement at the wrist in CP and the characteristic wrist position of such patients.
How to Talk About the Body?
The Normative Dimension of Science
Body & Society © 2004 SAGE Publications (London, Thousand Oaks and New Delhi),
Vol. 10(2–3): 205–229
During the conference that provided the occasion for this issue of Body &
Society, I did a little test and asked everyone to write down what the antonym of
the word ‘body’ was. In the long list I compiled, apart from predictable and
amusing definitions like ‘antibody’ or ‘nobody’ the most arresting for me were:
‘unaffected’ and ‘death’. If the opposite of being a body is dead, there is no life
to expect apart from the body, especially not an after-life, nor a life of a mind:
either you have, you are a body, or you are dead, you have become a corpse, you
enter into some sort of macabre body count. This is a direct consequence of
Vinciane Despret’s argument (in this issue) drawing on William James on
emotion: to have a body is to learn to be affected, meaning ‘effectuated’, moved,
put into motion by other entities, humans or non-humans. If you are not engaged
in this learning you become insensitive, dumb, you drop dead.